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According to Encyclopedia Brittanica, “....each photoreceptor cell contains a nucleus.... that conveys electrical signals to the next neurons in the processing chain.”
According to Doctor Bazan, the Director of the Neuroscience Centre of Excellence at LSU Health, New Orleans School of Medicine,
“…. the retina is key in age-related macular degeneration .... . As humans age, ... tissues become littered with dysfunctional cells, which then attract inflammatory immune cells. ... the resultant chronic inflammation can lead to brain and photoreceptor cell death.....”
So, Encyclopedia Brittanica tells us how rods and cons in our eyes function. And, Doctor Bazan confirms that tissues become littered with dysfunctional cells.
What Doctor Bazan still has to find out, maybe because he happens not to have this disease, is that these dysfunctional cells, in the retina, pile up.
And, the dysfunctional cells do not kill any photoreceptors, but damage some.
So, when we strain our eyes, we short circuit the conveying of electrical signals to the next neurons in the processing chain, producing excess rods and cons which become dysfunctional. These rods and cons pile up and cover the other normally working photoreceptors, and ultimately strangulate them.
And that, is what we refer to as vision loss.
I did not know it too, till my own crazy journey with dry macular degeneration which turned to wet.
With the help of a lot of research information, I was able to successfully reverse the effects of both dry and wet macular degeneration.
Meaning all those researchers and doctors can do it too.
So, if you suffer from macular degeneration, there is hope that you will be able to regain either part of your vision or all of it again.
Age triggers macular degeneration because of the period the rods and cons malfunction in people’s eyes is not cleaned out.
There are other triggers too, like the accumulated load of the malfunctioning rods and cons. These ‘rogue’ rods and cons sit on your RPE cells and their load damages the blood vessels they sit on, turning the dry form of this disease into the wet form.
That load then spreads along the RPE, depressing that layer, resulting in what doctors and researchers are calling the thinning of the macular.
So, the reason why all the interventions already on the market are failing to give desired results is because the approach to the macular problem is addressing the wrong concern.
The real concern is the ‘strangulation’ of the photoreceptors by these accumulated ‘rogue’ rods and cons.
That is what causes the gradual or rapid loss of vision.
The dent in the RPE scans, is caused by dysfunctional rods and cons sitting on it. So, vision loss in macular disease is caused by the total cover up and therefore ‘strangulation’ of the photoreceptors by ‘rogue’ rods and cons, a result of accumulated eye strain.
The best example I can give is for anyone to pile up a lot of small transparent discs and try to see through them. There will be distortion. That is, the same story with the macular disease.
The ‘rogue’ rods and cons, battle to let light through, confuse the brain because of distorted images they send through, and the brain in turn transduces what it is fed, resulting in our eyes ‘seeing’ grotesque or distorted images.
How these accumulated ‘rogue’ rods and cons arrange themselves also affects the way the eyes battle in compensating vision loss. If the ‘rogue’ rods and cons load is removed from the RPE cells, the resultant relief from pressure frees the photoreceptors to do their job.
I know this because I have successfully experienced it.
So, if your RPE cells are damaged, you should still be able to see well enough to lead an almost normal life again.
Thankfully, any damage to the RPE cells is not the ability to provide vision, but maybe a secondary and still to be accurately defined role.

Salud, mente y cuerpo
diciembre 7
Agnes Musa
Smashwords, Inc.

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